Reactive Oxygen Species-Mediated Loss of Synaptic Akt1 Signaling Leads to Deficient Activity-Dependent Protein Translation Early in Alzheimer's Disease-Reference-Cited by-同舟云学术

Reactive Oxygen Species-Mediated Loss of Synaptic Akt1 Signaling Leads to Deficient Activity-Dependent Protein Translation Early in Alzheimer's Disease

Published:2017-12 Issue:16 Volume:27 Page:1269-1280
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Ahmad Faraz¹,Singh Kunal¹,Das Debajyoti¹,Gowaikar Ruturaj¹,Shaw Eisha¹,Ramachandran Arathy¹,Rupanagudi Khader Valli¹,Kommaddi Reddy Peera¹,Bennett David A.²,Ravindranath Vijayalakshmi¹³

Affiliation:

1. Centre for Neuroscience, Indian Institute of Science, Bangalore, India.

2. Rush Alzheimer's Disease Center, Rush University Medical Center, Chicago, Illinois.

3. Centre for Brain Research, Bangalore, India.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6860

Reference50 articles.

1. Critical cysteines in Akt1 regulate its activity and proteasomal degradation: implications for neurodegenerative diseases

2. Culturing pyramidal neurons from the early postnatal mouse hippocampus and cortex

3. Overview and Findings from the Rush Memory and Aging Project

4. Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins

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