Type I Interferon Signaling is Required for Oncostatin-M Driven Inflammatory Responses in Mouse Lung
Author:
Affiliation:
1. Triumvera Biologics, Hamilton, Ontario, Canada.
2. Department of Medicine, McMaster Immunology Research Centre (MIRC), McMaster University, Hamilton, Ontario, Canada.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
https://www.liebertpub.com/doi/pdf/10.1089/jir.2022.0136
Reference38 articles.
1. Overexpression of OSM and IL-6 impacts the polarization of pro-fibrotic macrophages and the development of bleomycin-induced lung fibrosis
2. Pathogenicity of different PR8 influenza A virus variants in mice is determined by both viral and host factors
3. Pulmonary Expression of Oncostatin M (OSM) Promotes Inducible BALT Formation Independently of IL-6, Despite a Role for IL-6 in OSM-Driven Pulmonary Inflammation
4. Oncostatin M in the Regulation of Connective Tissue Cells and Macrophages in Pulmonary Disease
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