Overexpression of OSM and IL-6 impacts the polarization of pro-fibrotic macrophages and the development of bleomycin-induced lung fibrosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-13511-z.pdf
Reference62 articles.
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2. Gifford, A. H., Matsuoka, M., Ghoda, L. Y., Homer, R. J. & Enelow, R. I. Chronic inflammation and lung fibrosis: pleotropic syndromes but limited distinct phenotypes. Mucosal Immunol 5, 480–484, https://doi.org/10.1038/mi.2012.68 (2012).
3. Bringardner, B. D., Baran, C. P., Eubank, T. D. & Marsh, C. B. The role of inflammation in the pathogenesis of idiopathic pulmonary fibrosis. Antioxid Redox Signal 10, 287–301, https://doi.org/10.1089/ars.2007.1897 (2008).
4. Ask, K., Martin, G. E., Kolb, M. & Gauldie, J. Targeting genes for treatment in idiopathic pulmonary fibrosis: challenges and opportunities, promises and pitfalls. Proceedings of the American Thoracic Society 3, 389–393, https://doi.org/10.1513/pats.200602-021TK (2006).
5. Moeller, A., Ask, K., Warburton, D., Gauldie, J. & Kolb, M. The bleomycin animal model: a useful tool to investigate treatment options for idiopathic pulmonary fibrosis? The international journal of biochemistry & cell biology 40, 362–382, https://doi.org/10.1016/j.biocel.2007.08.011 (2008).
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