Overexpression of indoleamine 2,3-dioxygenase correlates with regulatory T cell phenotype in acute myeloid leukemia patients with normal karyotype
Author:
Affiliation:
1. Hematology Research Center, Shiraz, Iran.
2. Department of Pathology, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Publisher
The Korean Society of Hematology
Subject
Hematology
Link
https://synapse.koreamed.org/pdf/10.5045/br.2018.53.4.294
Reference26 articles.
1. Tryptophan catabolism and T-cell tolerance: immunosuppression by starvation?
2. Indoleamine 2,3-dioxygenase activity of acute myeloid leukemia cells can be measured from patients’ sera by HPLC and is inducible by IFN-γ
3. Tryptophan-derived Catabolites Are Responsible for Inhibition of T and Natural Killer Cell Proliferation Induced by Indoleamine 2,3-Dioxygenase
4. Potential Regulatory Function of Human Dendritic Cells Expressing Indoleamine 2,3-Dioxygenase
5. Human bone marrow stromal cells inhibit allogeneic T-cell responses by indoleamine 2,3-dioxygenase–mediated tryptophan degradation
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