Response by Xu et al to Letter Regarding Article, “Branched-Chain Amino Acid Catabolism Promotes Thrombosis Risk by Enhancing Tropomodulin-3 Propionylation in Platelets”-Reference-Cited by-同舟云学术

Response by Xu et al to Letter Regarding Article, “Branched-Chain Amino Acid Catabolism Promotes Thrombosis Risk by Enhancing Tropomodulin-3 Propionylation in Platelets”

Published:2020-12-15 Issue:24 Volume:142 Page:
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Xu Yanyan¹,Jiang Haojie¹,Liu Junling¹²

Affiliation:

1. Department of Biochemistry and Molecular Cell Biology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University School of Medicine, China (Y.X., H.J., J.L.).

2. The Cyrus Tang Hematology Center, Collaborative Innovation Center of Hematology, Soochow University, Suzhou, China (J.L.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference4 articles.

1. Branched-Chain Amino Acid Catabolism Promotes Thrombosis Risk by Enhancing Tropomodulin-3 Propionylation in Platelets

2. Differential regulation of mTORC1 by leucine and glutamine

3. Genetic Predisposition to an Impaired Metabolism of the Branched-Chain Amino Acids and Risk of Type 2 Diabetes: A Mendelian Randomisation Analysis

4. Branched-chain amino acid metabolism in chronic renal failure.;Tizianello A;Kidney Int Suppl,1983

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