Puma Deletion Delays Cardiac Dysfunction in Murine Heart Failure Models Through Attenuation of Apoptosis-Reference-Cited by-同舟云学术

Puma Deletion Delays Cardiac Dysfunction in Murine Heart Failure Models Through Attenuation of Apoptosis

Published:2011-07-05 Issue:1 Volume:124 Page:31-39
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Mandl Adel¹,Huong Pham Ly¹,Toth Kalman¹,Zambetti Gerald¹,Erhardt Peter¹

Affiliation:

1. From the Boston Biomedical Research Institute, Watertown, MA (A.M., L.H.P., P.E.); First Department of Medicine, Division of Cardiology, University of Pecs Medical School, Pecs, Hungary (A.M., K.T.); and Cancer Center, St. Jude Children's Research Hospital, Memphis, TX (G.Z.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference36 articles.

1. Cardiac Plasticity

2. Acute Doxorubicin Cardiotoxicity Is Associated With p53-Induced Inhibition of the Mammalian Target of Rapamycin Pathway

3. p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

4. p53 Activates the Mitochondrial Death Pathway and Apoptosis of Ventricular Myocytes Independent of de novo Gene Transcription

5. Lack of JunD Promotes Pressure Overload–Induced Apoptosis, Hypertrophic Growth, and Angiogenesis in the Heart

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