Potential Role of BNIP3 in Cardiac Remodeling, Myocardial Stiffness, and Endoplasmic Reticulum-Reference-Cited by-同舟云学术

Potential Role of BNIP3 in Cardiac Remodeling, Myocardial Stiffness, and Endoplasmic Reticulum

Published:2013-05 Issue:3 Volume:6 Page:572-583
ISSN:1941-3289
Container-title:Circulation: Heart Failure
language:en
Short-container-title:Circ Heart Fail

Author:

Chaanine Antoine H.¹,Gordon Ronald E.¹,Kohlbrenner Erik¹,Benard Ludovic¹,Jeong Dongtak¹,Hajjar Roger J.¹

Affiliation:

1. From the Cardiovascular Institute (A.H.C., E.K., L.B., D.J., R.J.H.), and Pathology Department (R.E.G.), Mount Sinai School of Medicine, New York, NY.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference27 articles.

1. Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice

2. Mitochondrial Pruning by Nix and BNip3: An Essential Function for Cardiac-Expressed Death Factors

3. A Novel Hypoxia-Inducible Spliced Variant of Mitochondrial Death Gene Bnip3 Promotes Survival of Ventricular Myocytes

4. Bnip3 functions as a mitochondrial sensor of oxidative stress during myocardial ischemia and reperfusion

5. The BH3‐only Bnip3 binds to the dynamin Opa1 to promote mitochondrial fragmentation and apoptosis by distinct mechanisms

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