Response by Good et al to Letter Regarding Article, “Pannexin-1 Channels as an Unexpected New Target of the Antihypertensive Drug Spironolactone”-Reference-Cited by-同舟云学术

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Response by Good et al to Letter Regarding Article, “Pannexin-1 Channels as an Unexpected New Target of the Antihypertensive Drug Spironolactone”

Published:2018-05-25 Issue:11 Volume:122 Page:
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Good Miranda E.¹,Chiu Yu-Hsin²,Poon Ivan K.H.³,Jaffe Iris Z.⁴,Bayliss Douglas A.²,Isakson Brant E.¹,Ravichandran Kodi S.⁵

Affiliation:

1. Department of Molecular Physiology and Biophysics, Robert M. Berne Cardiovascular Research Center, University of Virginia School of Medicine, Charlottesville

2. Department of Pharmacology, University of Virginia School of Medicine, Charlottesville

3. Department of Biochemistry and Genetics, La Trobe University, Melbourne, Australia

4. Molecular Cardiology Research Institute, Tufts Medical Center, Boston, MA

5. Department of Microbiology, Immunology, and Cancer Biology, University of Virginia School of Medicine, Charlottesville

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference13 articles.

1. Pannexin-1 channels do not regulate α1-adrenoceptor-mediated vasoconstriction in resistance arteries

2. ATP is not involved in α1-adrenoceptor-mediated vasoconstriction in resistance arteries

3. Pannexin1 Regulates α1-Adrenergic Receptor– Mediated Vasoconstriction

4. Pannexins, distant relatives of the connexin family with specific cellular functions?

5. Novel α1-adrenoceptor antagonism by the fluroquinolone antibiotic trovafloxacin

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