Positive Feedback Regulation of Agonist-Stimulated Endothelial Ca 2+ Dynamics by K Ca 3.1 Channels in Mouse Mesenteric Arteries-Reference-Cited by-同舟云学术

Positive Feedback Regulation of Agonist-Stimulated Endothelial Ca 2+ Dynamics by K Ca 3.1 Channels in Mouse Mesenteric Arteries

Published:2014-01 Issue:1 Volume:34 Page:127-135
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Qian Xun¹,Francis Michael¹,Köhler Ralf¹,Solodushko Viktoriya¹,Lin Mike¹,Taylor Mark S.¹

Affiliation:

1. From the Department of Physiology, University of South Alabama College of Medicine, Mobile, AL (X.Q., M.F., V.S., M.L., M.S.T.); Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark (R.K.); and Translational Research Unit, University Hospital Miguel Servet, Aragon Institute of Health Sciences and ARAID, Zaragoza, Spain (R.K.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference38 articles.

1. EDHF is not K+ but may be due to spread of current from the endothelium in guinea pig arterioles

2. Characterization of an apamin-sensitive small-conductance Ca2+ -activated K+ channel in porcine coronary artery endothelium: relevance to EDHF

3. Characterization of a charybdotoxin-sensitive intermediate conductance Ca2+ -activated K+ channel in porcine coronary endothelium: relevance to EDHF

4. Myoendothelial Gap Junctions May Provide the Pathway for EDHF in Mouse Mesenteric Artery

5. Altered Expression of Small-Conductance Ca 2+ -Activated K + (SK3) Channels Modulates Arterial Tone and Blood Pressure

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