Role of Angiotensin II and Reactive Oxygen Species in Cyclosporine A–Dependent Hypertension-Reference-Cited by-同舟云学术

Role of Angiotensin II and Reactive Oxygen Species in Cyclosporine A–Dependent Hypertension

Published:2003-10 Issue:4 Volume:42 Page:754-760
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Nishiyama Akira¹,Kobori Hiroyuki¹,Fukui Toshiki¹,Zhang Guo-Xing¹,Yao Li¹,Rahman Matlubur¹,Hitomi Hirofumi¹,Kiyomoto Hideyasu¹,Shokoji Takatomi¹,Kimura Shoji¹,Kohno Masakazu¹,Abe Youichi¹

Affiliation:

1. From the Department of Pharmacology (A.N., G.-X.Z, L.Y., M.R., H.H., S.K., Y.A.) and the Second Department of Internal Medicine (T.F., H.Kiyomoto, T.S., M.K.), Kagawa Medical University, Kagawa, Japan; and the Department of Physiology (H.Kobori), Tulane University Health Sciences Center, New Orleans, La.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference47 articles.

1. Cyclosporin

2. Hypertensinogenic mechanism of the calcineurin inhibitors

3. Interaction of Cyclosporine A and the Renin-Angiotensin System New Perspectives

4. Effect of CsA on the expression of renin and angiotensin type 1 receptor genes in the rat kidney

Cited by 99 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Chenodeoxycholic acid alleviated the cyclosporine-induced nephrotoxicity by decreasing oxidative stress and suppressing renin-angiotensin system through AT2R and ACE2 mRNA upregulation in rats;2024-07-22

2. Minimizing Metabolic and Cardiac Risk Factors to Maximize Outcomes After Liver Transplantation;Transplantation;2023-12-07

3. Phosphatase-independent activity of smooth-muscle calcineurin orchestrates a gene expression program leading to hypertension;2023-11-26

4. Calcineurin Is a Universal Regulator of Vessel Function—Focus on Vascular Smooth Muscle Cells;Cells;2023-09-13

5. The effect of glycine on oxidative stress, inflammation and renin-angiotensin system in kidneys and aorta of cyclosporine-administered rats;Drug and Chemical Toxicology;2023-06-20