Recruitment of RNA Polymerase II to Metabolic Gene Promoters Is Inhibited in the Failing Heart Possibly Through PGC-1α (Peroxisome Proliferator-Activated Receptor-γ Coactivator-1α) Dysregulation-Reference-Cited by-同舟云学术

Recruitment of RNA Polymerase II to Metabolic Gene Promoters Is Inhibited in the Failing Heart Possibly Through PGC-1α (Peroxisome Proliferator-Activated Receptor-γ Coactivator-1α) Dysregulation

Published:2019-03 Issue:3 Volume:12 Page:
ISSN:1941-3289
Container-title:Circulation: Heart Failure
language:en
Short-container-title:Circ: Heart Failure

Author:

Bhat Santosh¹,Chin Adave¹,Shirakabe Akihiro¹,Ikeda Yoshiyuki¹,Ikeda Shohei¹,Zhai Peiyong¹,Hsu Chiao-po²,Sayed Danish¹,Abdellatif Maha¹,Byun Jaemin¹,Schesing Kevin¹,Tang Fan¹,Tian Yimin¹,Babu Gopal¹,Ralda Guersom¹,Warren Junco S.³,Cho Jaeyeaon⁴,Sadoshima Junichi¹,Oka Shin-ichi¹

Affiliation:

1. Department of Cell Biology and Molecular Medicine, Rutgers Biomedical Health Sciences, Newark, NJ (S.B., A.C., A.S., Y.I., S.I., P.Z., D.S., M.A., J.B., K.S., F.T., Y.T., G.B., G.R., J.S., S.-i.O.).

2. Division of Cardiovascular Surgery, Department of Surgery, Veterans General Hospital, National Yang-Ming University School of Medicine, Taiwan (C.-p.H.).

3. Nora Eccles Harrison Cardiovascular Research and Training Institute, Department of Internal Medicine, University of Utah, Salt Lake City (J.S.W.).

4. Department of Biomedical Science, College of Medicine, Avison Biomedical Research Center, Yonsei University College of Medicine, Seoul, Korea (J.C.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference17 articles.

1. PGC-1α and ERRα target gene downregulation is a signature of the failing human heart

2. Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR- coactivator 1

3. PGC-1α Regulates Expression of Myocardial Mitochondrial Antioxidants and Myocardial Oxidative Stress After Chronic Systolic Overload

4. Disruption of Sarcolemmal ATP-Sensitive Potassium Channel Activity Impairs the Cardiac Response to Systolic Overload

5. Promoting PGC-1α-driven mitochondrial biogenesis is detrimental in pressure-overloaded mouse hearts

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