PGC-1α Regulates Expression of Myocardial Mitochondrial Antioxidants and Myocardial Oxidative Stress After Chronic Systolic Overload-Reference-Cited by-同舟云学术

PGC-1α Regulates Expression of Myocardial Mitochondrial Antioxidants and Myocardial Oxidative Stress After Chronic Systolic Overload

Published:2010-10 Issue:7 Volume:13 Page:1011-1022
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Lu Zhongbing¹²³,Xu Xin¹²,Hu Xinli¹²,Fassett John¹,Zhu Guangshuo¹,Tao Yi¹³,Li Jingxin⁴,Huang Yimin⁴,Zhang Ping¹,Zhao Baolu³,Chen Yingjie¹²

Affiliation:

1. Cardiovascular Division, University of Minnesota, Minneapolis, Minnesota.

2. Lillehei Heart Institute, University of Minnesota, Minneapolis, Minnesota.

3. State Key Laboratory of Brain and Recognition Laboratory, Institute of Biophysics, The Chinese Academy of Sciences, Beijing, China.

4. Beijing Anzhen Hospital, Capital Medical University, Anzhenli, Chaoyang District, Beijing, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2940

Reference34 articles.

1. Transcriptional coactivator PGC-1α controls the energy state and contractile function of cardiac muscle

2. Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR- coactivator 1

3. Mitochondria, Oxidants, and Aging

4. Essential Role for Mitochondrial Thioredoxin Reductase in Hematopoiesis, Heart Development, and Heart Function

5. Peroxisome Proliferator–Activated Receptor γ Coactivator-1 (PGC-1) Regulatory Cascade in Cardiac Physiology and Disease

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