“La Donna è Mobile…”
Author:
Affiliation:
1. From the Unit of Pharmacology and Therapeutics (FATH 5349), Department of Medicine, Université catholique de Louvain, Brussels, Belgium.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Reference30 articles.
1. Control of cardiac muscle cell function by an endogenous nitric oxide signaling system.
2. A Defect of Neuronal Nitric Oxide Synthase Increases Xanthine Oxidase-Derived Superoxide Anion and Attenuates the Control of Myocardial Oxygen Consumption by Nitric Oxide Derived From Endothelial Nitric Oxide Synthase
3. Endogenous nitric oxide mechanisms mediate the stretch dependence of Ca2+ release in cardiomyocytes
4. Cardiomyocyte-Specific Overexpression of Nitric Oxide Synthase 3 Improves Left Ventricular Performance and Reduces Compensatory Hypertrophy After Myocardial Infarction
5. Nitric Oxide and Cardiac Function
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1. The Endothelium, Cardiovascular Disease, and Therapy;Metabolic Syndrome and Cardiovascular Disease;2012-08-06
2. Skeletal Muscle and Exercise;Metabolic Syndrome and Cardiovascular Disease;2012-08-06
3. Displacement-encoded and manganese-enhanced cardiac MRI reveal that nNOS, not eNOS, plays a dominant role in modulating contraction and calcium influx in the mammalian heart;American Journal of Physiology-Heart and Circulatory Physiology;2012-01
4. Endothelial nitric oxide synthase Glu298Asp gene polymorphism in a multi-ethnical population with heart failure and controls;Nitric Oxide;2010-04
5. eNOS Activation by Physical Forces: From Short-Term Regulation of Contraction to Chronic Remodeling of Cardiovascular Tissues;Physiological Reviews;2009-04
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