Resolving Thromboinflammation in the Brain After Ischemic Stroke?-Reference-Cited by-同舟云学术

Resolving Thromboinflammation in the Brain After Ischemic Stroke?

Published:2016-05-31 Issue:22 Volume:133 Page:2128-2131
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Kehrel Beate E.¹,Fender Anke C.¹

Affiliation:

1. From Department of Anaesthesiology, Intensive Care and Pain Medicine, Experimental and Clinical Haemostasis Research Group, University Hospital Muenster, Germany (B.E.K.); and Institute of Pharmacology and Clinical Pharmacology, Heinrich-Heine-University, Duesseldorf, Germany (A.C.F.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference18 articles.

1. Thromboinflammation in Stroke Brain Damage

2. Unwinding the von Willebrand factor strings puzzle

3. ADAMTS13 and von Willebrand Factor in Thrombotic Thrombocytopenic Purpura

4. Low ADAMTS13 activity is associated with an increased risk of ischemic stroke

5. Glycoprotein VI is a major collagen receptor for platelet activation: it recognizes the platelet-activating quaternary structure of collagen, whereas CD36, glycoprotein IIb/IIIa, and von Willebrand factor do not.;Kehrel B;Blood,1998

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