Letter by London Regarding Article, “Reappraisal of Reported Genes for Sudden Arrhythmic Death: Evidence-Based Evaluation of Gene Validity for Brugada Syndrome”
Author:
Affiliation:
1. Division of Cardiovascular Medicine and Abboud Cardiovascular Research Center, University of Iowa Carver College of Medicine, Iowa City.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Reference5 articles.
1. Mutation in Glycerol-3-Phosphate Dehydrogenase 1–Like Gene ( GPD1-L ) Decreases Cardiac Na + Current and Causes Inherited Arrhythmias
2. Molecular and Functional Characterization of Novel Glycerol-3-Phosphate Dehydrogenase 1–Like Gene ( GPD1-L ) Mutations in Sudden Infant Death Syndrome
3. Reappraisal of Reported Genes for Sudden Arrhythmic Death
4. Emerging potential benefits of modulating NAD+ metabolism in cardiovascular disease
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1. Estudio electrofisiológico como método de estratificación de riesgo de muerte súbita en el Síndrome de Brugada;Tesla Revista Científica;2023-05-24
2. Brugada Syndrome: From Molecular Mechanisms and Genetics to Risk Stratification;International Journal of Molecular Sciences;2023-02-07
3. The Mechanism of Ajmaline and Thus Brugada Syndrome: Not Only the Sodium Channel!;Frontiers in Cardiovascular Medicine;2021-12-23
4. Commentary: Peptide-Based Targeting of the L-Type Calcium Channel Corrects the Loss-of-Function Phenotype of Two Novel Mutations of the CACNA1 Gene Associated With Brugada Syndrome;Frontiers in Physiology;2021-06-09
5. Evaluating the Use of Genetics in Brugada Syndrome Risk Stratification;Frontiers in Cardiovascular Medicine;2021-04-21
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