Cellular Mechanism of Vasoconstriction Induced by Angiotensin II-Reference-Cited by-同舟云学术

Cellular Mechanism of Vasoconstriction Induced by Angiotensin II

Published:2003-11-28 Issue:11 Volume:93 Page:1015-1017
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Kanaide Hideo¹,Ichiki Toshihiro¹,Nishimura Junji¹,Hirano Katsuya¹

Affiliation:

1. From the Division of Molecular Cardiology (H.K., J.N., K.H.) and Cardiovascular Medicine (T.I.), Research Institute of Angiocardiology, Graduate School of Medical Sciences, and Kyushu University COE Program on Lifestyle-Related Diseases, Kyushu University, Fukuoka, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Link

https://www.ahajournals.org/doi/pdf/10.1161/01.RES.0000105920.33926.60

Reference19 articles.

1. AT1b Receptor Predominantly Mediates Contractions in Major Mouse Blood Vessels

2. New Roads Leading to Ca 2+ Sensitization

3. Protein kinase C in the regulation of smooth muscle contraction

4. Phosphatidylcholine is a major source of phosphatidic acid and diacylglycerol in angiotensin II-stimulated vascular smooth-muscle cells

5. Changes in the cytosolic Ca2+concentration and Ca2+-sensitivity of the contractile apparatus during angiotensin II-induced desensitization in the rabbit femoral artery

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