Neurogenic Mechanisms Contribute to Hypertension in Mice With Disruption of the K-Cl Cotransporter KCC3-Reference-Cited by-同舟云学术

Neurogenic Mechanisms Contribute to Hypertension in Mice With Disruption of the K-Cl Cotransporter KCC3

Published:2006-03-03 Issue:4 Volume:98 Page:549-556
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Rust Marco B.¹,Faulhaber Jörg¹,Budack Mareike K.¹,Pfeffer Carsten¹,Maritzen Tanja¹,Didié Michael¹,Beck Franz-Xaver¹,Boettger Thomas¹,Schubert Rudolf¹,Ehmke Heimo¹,Jentsch Thomas J.¹,Hübner Christian A.¹

Affiliation:

1. From the Zentrum für Molekulare Neurobiologie Hamburg (M.B.R., C.P., T.M., T.B., T.J.J., C.A.H.), Universität Hamburg; Institut für Vegetative Physiologie und Pathophysiologie (J.F., M.K.B., H.E.), Institut für Experimentelle und Klinische Pharmakologie (M.D.), and Institut für Humangenetik (C.A.H.), Universitätsklinikum Hamburg-Eppendorf, Hamburg; Physiologisches Institut (F.-X.B.), Ludwig-Maximilians-Universität München; and Institut für Physiologie (R.S.), Universität Rostock, Germany....

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference30 articles.

1. The Andermann Syndrome: Agenesis of the Corpus Callosum Associated with Mental Retardation and Progressive Sensorimotor Neuronopathy

2. The K–Cl cotransporter KCC3 is mutant in a severe peripheral neuropathy associated with agenesis of the corpus callosum

3. Loss of K-Cl co-transporter KCC3 causes deafness, neurodegeneration and reduced seizure threshold

4. Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4

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