Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/416874a.pdf
Reference30 articles.
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2. Kubisch, C. et al. KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness. Cell 96, 437–446 (1999).
3. Kharkovets, T. et al. KCNQ4, a K+ channel mutated in a form of dominant deafness, is expressed in the inner ear and the central auditory pathway. Proc. Natl Acad. Sci. USA 97, 4333–4338 (2000).
4. Kikuchi, T., Kimura, R. S., Paul, D. L., Takasaka, T. & Adams, J. C. Gap junction systems in the mammalian cochlea. Brain Res. Brain Res. Rev. 32, 163–166 (2000).
5. Jentsch, T. J. Neuronal KCNQ potassium channels: physiology and role in disease. Nature Rev. Neurosci. 1, 21–30 (2000).
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