Preeclampsia Pathogenesis
Author:
Affiliation:
1. From the Beth Israel Deaconess Medical Center and Harvard Medical School (S.A.K.), Boston, Mass; and the University of Chicago (M.D.L.), Ill.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Internal Medicine
Link
https://www.ahajournals.org/doi/pdf/10.1161/HYPERTENSIONAHA.107.100735
Reference18 articles.
1. Soluble Endoglin and Other Circulating Antiangiogenic Factors in Preeclampsia
2. Soluble endoglin contributes to the pathogenesis of preeclampsia
3. Placental ischemia and soluble fms-like tyrosine kinase 1: Cause or consequence of preeclampsia?
4. Negative Regulation of Soluble Flt-1 and Soluble Endoglin Release by Heme Oxygenase-1
5. Increased expression of sFlt-1 in in vivo and in vitro models of human placental hypoxia is mediated by HIF-1
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4. Comparison of serum soluble edoglin (sEng) level in eary onset preeclampsia, late onset preeclampsia and normal pregnant woman;Majalah Obstetri & Ginekologi;2018-03-31
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