Soluble endoglin contributes to the pathogenesis of preeclampsia-Reference-Cited by-同舟云学术

Soluble endoglin contributes to the pathogenesis of preeclampsia

Published:2006-06-01 Issue:6 Volume:12 Page:642-649
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Venkatesha Shivalingappa,Toporsian Mourad,Lam Chun,Hanai Jun-ichi,Mammoto Tadanori,Kim Yeon M,Bdolah Yuval,Lim Kee-Hak,Yuan Hai-Tao,Libermann Towia A,Stillman Isaac E,Roberts Drucilla,D'Amore Patricia A,Epstein Franklin H,Sellke Frank W,Romero Roberto,Sukhatme Vikas P,Letarte Michelle,Karumanchi S Ananth

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1429.pdf

Reference50 articles.

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3. Roberts, J.M. et al. Preeclampsia: an endothelial cell disorder. Am. J. Obstet. Gynecol. 161, 1200–1204 (1989).

4. Maynard, S.E. et al. Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. J. Clin. Invest. 111, 649–658 (2003).

5. Zhou, Y. et al. Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets syndrome. Am. J. Pathol. 160, 1405–1423 (2002).

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