Syndecan-1 Amplifies Angiotensin II–Induced Cardiac Fibrosis-Reference-Cited by-同舟云学术

Syndecan-1 Amplifies Angiotensin II–Induced Cardiac Fibrosis

Published:2010-02 Issue:2 Volume:55 Page:249-256
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Schellings Mark W.M.¹,Vanhoutte Davy¹,van Almen Geert C.¹,Swinnen Melissa¹,Leenders Joost J.G.¹,Kubben Nard¹,van Leeuwen Rick E.W.¹,Hofstra Leo¹,Heymans Stephane¹,Pinto Yigal M.¹

Affiliation:

1. From the Heart Failure Research Center (M.W.M.S., G.C.v.A., M.S., N.K., R.E.W.v.L., S.H.) and Department of Cardiology (M.W.M.S., L.H., S.H.), Cardiovascular Research Institute Maastricht, University Hospital Maastricht, Maastricht, The Netherlands; Vesalius Research Center (D.V., M.S.), Flemish Institute for Biotechnology-Catholic University Leuven, Leuven, Belgium; Heart Failure Research Center (J.J.G.L., Y.M.P.), Amsterdam Medical Center, Amsterdam, The Netherlands.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference24 articles.

1. Apoptosis Signal-Regulating Kinase 1 Plays a Pivotal Role in Angiotensin II–Induced Cardiac Hypertrophy and Remodeling

2. Extracellular signal regulated kinase and SMAD signaling both mediate the angiotensin II driven progression towards overt heart failure in homozygous TGR(mRen2)27

3. Connective Tissue Growth Factor Is a Mediator of Angiotensin II–Induced Fibrosis

4. Transforming Growth Factor β Receptor Endoglin Is Expressed in Cardiac Fibroblasts and Modulates Profibrogenic Actions of Angiotensin II

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