Fluid Shear Stress Activates Proline-Rich Tyrosine Kinase via Reactive Oxygen Species–Dependent Pathway-Reference-Cited by-同舟云学术

Fluid Shear Stress Activates Proline-Rich Tyrosine Kinase via Reactive Oxygen Species–Dependent Pathway

Published:2002-11 Issue:11 Volume:22 Page:1790-1796
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Tai Lung-Kuo¹,Okuda Masanori¹,Abe Jun-ichi¹,Yan Chen¹,Berk Bradford C.¹

Affiliation:

1. From the Center for Cardiovascular Research (L.-K.T., J.A., C.Y., B.C.B.), University of Rochester, Rochester, NY, and the Division of Cardiovascular and Respiratory Medicine (M.O.), Kobe University Graduate School of Medicine, Kobe, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference45 articles.

1. Flow-mediated endothelial mechanotransduction

2. Shear-induced platelet-derived growth factor gene expression in human endothelial cells is mediated by protein kinase C

3. Fluid shear stress induces a biphasic response of human monocyte chemotactic protein 1 gene expression in vascular endothelium.

4. Fluid shear stress activation of egr-1 transcription in cultured human endothelial and epithelial cells is mediated via the extracellular signal-related kinase 1/2 mitogen-activated protein kinase pathway.

5. The Ras-JNK pathway is involved in shear-induced gene expression

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