Activation of Nod-Like Receptor Protein 3 Inflammasomes Turns on Podocyte Injury and Glomerular Sclerosis in Hyperhomocysteinemia-Reference-Cited by-同舟云学术

Activation of Nod-Like Receptor Protein 3 Inflammasomes Turns on Podocyte Injury and Glomerular Sclerosis in Hyperhomocysteinemia

Published:2012-07 Issue:1 Volume:60 Page:154-162
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Zhang Chun¹,Boini Krishna M.¹,Xia Min¹,Abais Justine M.¹,Li Xiang¹,Liu Qinglian¹,Li Pin-Lan¹

Affiliation:

1. From the Departments of Pharmacology and Toxicology (C.Z., K.M.B., M.X., J.M.A., X.L., P.-L.L.) and Physiology and Biophysics (Q.L.), Medical College of Virginia, Virginia Commonwealth University, Richmond, VA; current address (C.Z.): Department of Nephrology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference28 articles.

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4. Necrotic cells trigger a sterile inflammatory response through the Nlrp3 inflammasome

5. Podocytes are the major source of IL-1α and IL-1β in human glomerulonephritides

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