Acute Exertion Elicits a H 2 O 2 -Dependent Vasodilator Mechanism in the Microvasculature of Exercise-Trained but Not Sedentary Adults-Reference-Cited by-同舟云学术

Acute Exertion Elicits a H 2 O 2 -Dependent Vasodilator Mechanism in the Microvasculature of Exercise-Trained but Not Sedentary Adults

Published:2015-01 Issue:1 Volume:65 Page:140-145
ISSN:0194-911X
Container-title:Hypertension
language:en
Short-container-title:Hypertension

Author:

Durand Matthew J.¹,Dharmashankar Kodlipet¹,Bian Jing-Tan¹,Das Emon¹,Vidovich Mladen¹,Gutterman David D.¹,Phillips Shane A.¹

Affiliation:

1. From the Department of Medicine, Cardiovascular Center, Medical College of Wisconsin, Milwaukee (M.J.D., K.D., E.D., D.D.G.); and Department of Physical Therapy (J.-T.B., S.A.P.) and Department of Medicine, Cardiology (M.V., S.A.P.), University of Illinois at Chicago.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Internal Medicine

Reference24 articles.

1. Resistance and aerobic exercise protects against acute endothelial impairment induced by a single exposure to hypertension during exertion

2. The Effect of Exertional Hypertension Evoked by Weight Lifting on Vascular Endothelial Function

3. Arterial blood pressure response to heavy resistance exercise

4. Guidelines for the ultrasound assessment of endothelial-dependent flow-mediated vasodilation of the brachial artery

5. Resistance and aerobic exercise protects against acute endothelial impairment induced by a single exposure to hypertension during exertion

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