Delineating RBM20 Regulation of Alternative Splicing in Dilated Cardiomyopathy-Reference-Cited by-同舟云学术

Delineating RBM20 Regulation of Alternative Splicing in Dilated Cardiomyopathy

Published:2014-10 Issue:5 Volume:7 Page:732-733
ISSN:1942-325X
Container-title:Circulation: Cardiovascular Genetics
language:en
Short-container-title:Circ Cardiovasc Genet

Author:

Arrell D. Kent¹

Affiliation:

1. From the Center for Regenerative Medicine, Marriott Heart Disease Research Program, Division of Cardiovascular Diseases, Department of Medicine, and Department of Molecular Pharmacology and Experimental Therapeutics Mayo Clinic, Rochester, MN.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Genetics(clinical),Cardiology and Cardiovascular Medicine,Genetics

Link

https://www.ahajournals.org/doi/pdf/10.1161/CIRCGENETICS.114.000879

Reference9 articles.

1. Genetic mutations and mechanisms in dilated cardiomyopathy

2. RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing

3. Mutations in Ribonucleic Acid Binding Protein Gene Cause Familial Dilated Cardiomyopathy

4. Genetic variation in the alternative splicing regulator RBM20 is associated with dilated cardiomyopathy

5. Pathophysiological Defects and Transcriptional Profiling in the RBM20-/- Rat Model

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1. Dysregulated ribonucleoprotein granules promote cardiomyopathy in RBM20 gene-edited pigs;Nature Medicine;2020-11

2. Towards understanding pre-mRNA splicing mechanisms and the role of SR proteins;Gene;2016-08

3. New Insights Into the Role of RNA-Binding Proteins in the Regulation of Heart Development;International Review of Cell and Molecular Biology;2016