Duchenne Muscular Dystrophy Mice and Men
Author:
Affiliation:
1. From the Department of Physiology and Cell Biology (J.A.R.-F., J.A.C.), Division of Cardiovascular Medicine, Department of Internal Medicine (S.V.R.), College of Medicine and Wexner Medical Center, The Ohio State University, Columbus.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Reference18 articles.
1. Evolution of the mdx mouse cardiomyopathy: physiological and morphological findings
2. Skeletal muscle-specific expression of a utrophin transgene rescues utrophin-dystrophin deficient mice
3. Early Treatment With Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice
4. Utrophin deficiency worsens cardiac contractile dysfunction present in dystrophin-deficient mdx mice
5. Cardiomyopathic features associated with muscular dystrophy are independent of dystrophin absence in cardiovasculature
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