Granulocyte Colony Stimulating Factor Directly Inhibits Myocardial Ischemia-Reperfusion Injury Through Akt–Endothelial NO Synthase Pathway
Author:
Affiliation:
1. From the Department of Cardiovascular Science and Medicine, Chiba University Graduate School of Medicine, Japan.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine
Reference13 articles.
1. Postconditioning A new link in nature’s armor against myocardial ischemia–reperfusion injury
2. Postconditioning: A Form of “Modified Reperfusion” Protects the Myocardium by Activating the Phosphatidylinositol 3-Kinase-Akt Pathway
3. Mobilized bone marrow cells repair the infarcted heart, improving function and survival
4. Cytokine therapy prevents left ventricular remodeling and dysfunction after myocardial infarction through neovascularization
5. Acceleration of the Healing Process and Myocardial Regeneration May Be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Postinfarction Granulocyte Colony–Stimulating Factor Treatment
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