Modification of Endothelial NO Synthase Through Protein Phosphorylation After Forebrain Cerebral Ischemia/Reperfusion-Reference-Cited by-同舟云学术

Modification of Endothelial NO Synthase Through Protein Phosphorylation After Forebrain Cerebral Ischemia/Reperfusion

Published:2004-11 Issue:11 Volume:35 Page:2582-2586
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Osuka Koji¹,Watanabe Yasuo¹,Usuda Nobuteru¹,Nakazawa Ayami¹,Tokuda Masaaki¹,Yoshida Jun¹

Affiliation:

1. From the Department of Neurosurgery (K.O., J.Y.), Nagoya University Graduate School of Medicine, Japan; the Department of Cell Physiology (Y.W., M.T.), Kagawa University Faculty of Medicine, Japan; and the Department of Anatomy II (N.U., A.N.), Fujita Health University School of Medicine, Aichi, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference39 articles.

1. Nitric oxide, the enigmatic neuronal messenger: its role in synaptic plasticity

2. Bright and dark sides of nitric oxide in ischemic brain injury

3. Suppression of Apoptosis by Nitric Oxide via Inhibition of Interleukin-1β–converting Enzyme (ICE)-like and Cysteine Protease Protein (CPP)-32–like Proteases

4. Nitric Oxide Down-regulates MKP-3 mRNA Levels

5. Effects of Cerebral Ischemia in Mice Deficient in Neuronal Nitric Oxide Synthase

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