Activation of Protein Kinases in Chronically Hypoxic Infant Human and Rabbit Hearts-Reference-Cited by-同舟云学术

Activation of Protein Kinases in Chronically Hypoxic Infant Human and Rabbit Hearts

Published:2002-07-09 Issue:2 Volume:106 Page:239-245
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Rafiee Parvaneh¹,Shi Yang¹,Kong Xiangrong¹,Pritchard Kirkwood A.¹,Tweddell James S.¹,Litwin S. Bert¹,Mussatto Kathleen¹,Jaquiss Robert D.¹,Su Jidong¹,Baker John E.¹

Affiliation:

1. From the Divisions of Pediatric Surgery (P.R., Y.S., X.K., K.A.P., J.S., J.E.B.) and Cardiothoracic Surgery (J.S.T., S.B.L., R.D.J.), Medical College of Wisconsin, Milwaukee; and the Section of Cardiothoracic Surgery (J.S.T., S.B.L., K.M., R.D.J.), Children’s Hospital of Wisconsin, Milwaukee.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference22 articles.

1. Hypoxia alters the subcellular distribution of protein kinase C isoforms in neonatal rat ventricular myocytes.

2. Activation of c-Jun N-Terminal Kinases and p38-Mitogen-activated Protein Kinases in Human Heart Failure Secondary to Ischaemic Heart Disease

3. Chronic myocardial hypoxia increases nitric oxide synthase and decreases caveolin-3

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