Chronic myocardial hypoxia increases nitric oxide synthase and decreases caveolin-3

Author:

Shi Yang,Pritchard Kirkwood A,Holman Patricia,Rafiee Parvaneh,Griffith Owen W,Kalyanaraman B,Baker John E

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference22 articles.

1. Tolerance of the developing heart to ischemia;Baker;Am. J. Physiol.,1995

2. Baker, J. E.; Holman, P.; Kalyanaraman, B.; Griffith, O. W.; Pritchard, K. A. Jr. Adaptation to chronic hypoxia confers tolerance to subsequent myocardial ischemia by increased nitric oxide production. In: Cullinan, J.; Brennan, M. K., eds. Heart in stress. New York: The New York Academy of Science; 1999:235–253.

3. Prolonged in vivo hypoxia enhances nitric oxide synthase type I and type II gene expression in adult rat lung;Shaul;Am. J. Respir. Cell. Mol. Biol.,1995

4. Selective upregulation of arterial endothelial nitric oxide synthase in pulmonary hypertension;Resta;Am. J. Physiol.,1997

5. Chronic hypoxia upregulates endothelial and inducible NO synthase gene and protein expression in rat lung;Le Cras;Am. J. Physiol.,1996

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