Physiology and Pathophysiology of Vascular Signaling Controlled by Cyclic Guanosine 3′,5′-Cyclic Monophosphate–Dependent Protein Kinase-Reference-Cited by-同舟云学术

Physiology and Pathophysiology of Vascular Signaling Controlled by Cyclic Guanosine 3′,5′-Cyclic Monophosphate–Dependent Protein Kinase

Published:2003-11-04 Issue:18 Volume:108 Page:2172-2183
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Münzel Thomas¹,Feil Robert¹,Mülsch Alexander¹,Lohmann Suzanne M.¹,Hofmann Franz¹,Walter Ulrich¹

Affiliation:

1. From the Division of Cardiology, University Hospital Eppendorf, Hamburg, Germany (T.M.); Institut für Pharmakologie und Toxikologie, Technische Universität, München, Germany (R.F., F.H.); the Institute of Cardiovascular Physiology, J.W. Goethe–University Clinic, Frankfurt/Main, Germany (A.M.); and the Institute of Clinical Biochemistry and Pathobiochemistry, University of Würzburg, Würzburg, Germany (S.M.L., U.W.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference144 articles.

1. Prospects for Cardiovascular Research

2. The Guanylyl Cyclase Family at Y2K

3. Cardiac natriuretic peptides - hope or hype?

4. Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A–deficient mice

5. Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A

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