Activation of Kir2.1 improves myocardial fibrosis by inhibiting Ca <sup>2+</sup> overload and the TGF-β1/Smad signaling pathway-Reference-Cited by-同舟云学术

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Activation of Kir2.1 improves myocardial fibrosis by inhibiting Ca <sup>2+</sup> overload and the TGF-β1/Smad signaling pathway

Published:2023-05-01 Issue:5 Volume:55 Page:749-757
ISSN:1672-9145
Container-title:Acta Biochimica et Biophysica Sinica
language:en
Short-container-title:ABBS

Author:

Rong Yi,Zhou Xin,Guo Zhenli,Zhang Yingying,Qin Wenjuan,Li Li,Si Junqiang,Yang Rui,Li Xinzhi,Ma Ketao

Publisher

China Science Publishing & Media Ltd.

Subject

General Medicine,Biochemistry,Biophysics,Molecular Biology

Link

https://engine.scichina.com/doi/pdf/19D961C603CC4FBBB445E9F5668DF008

Reference29 articles.

1. Zhang W, Xu W, Feng Y, Zhou X. Non‐coding RNA involvement in the pathogenesis of diabetic cardiomyopathy. J Cell Mol Med, 2019, 23: 5859-5867.

2. Ma ZG, Yuan YP, Wu HM, Zhang X, Tang QZ. Cardiac fibrosis: new insights into the pathogenesis. Int J Biol Sci, 2018, 14: 1645-1657.

3. Yuan H, Fan Y, Wang Y, Gao T, Shao Y, Zhao B, Li H. Calcium‑sensing receptor promotes high glucose‑induced myocardial fibrosis via upregulation of the TGF‑β1/Smads pathway in cardiac fibroblasts. Mol Med Report, 2019, 20: 1093-1102.

4. Lindner D, Zietsch C, Tank J, Sossalla S, Fluschnik N, Hinrichs S, Maier L. Cardiac fibroblasts support cardiac inflammation in heart failure. Basic Res Cardiol, 2014, 109:

5. Stewart L, Turner NA. Channelling the force to reprogram the matrix: mechanosensitive ion channels in cardiac fibroblasts. Cells, 2021, 10:

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1. Identification of important genes related to anoikis in acute myocardial infarction;Journal of Cellular and Molecular Medicine;2024-03-25

2. Puerarin inhibits NHE1 activity by interfering with the p38 pathway and attenuates mitochondrial damage induced by myocardial calcium overload in heart failure rats;Acta Biochimica et Biophysica Sinica;2024-01-01

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