Puerarin inhibits NHE1 activity by interfering with the p38 pathway and attenuates mitochondrial damage induced by myocardial calcium overload in heart failure rats
Author:
Publisher
China Science Publishing & Media Ltd.
Subject
General Medicine,Biochemistry,Biophysics,Molecular Biology
Link
https://engine.scichina.com/doi/pdf/DA019BD1AB1A42009E305A8CD7D8A7C4
Reference61 articles.
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2. Zhang J, Wang Y, Bao C, Liu T, Li S, Huang J, Wan Y. Curcumin‑loaded PEG‑PDLLA nanoparticles for attenuating palmitate‑induced oxidative stress and cardiomyocyte apoptosis through AMPK pathway. Int J Mol Med, 2019, 44:
3. He Y, Huang W, Zhang C, Chen L, Xu R, Li N, Wang F. Energy metabolism disorders and potential therapeutic drugs in heart failure. Acta Pharmaceutica Sin B, 2021, 11: 1098-1116.
4. Ziaeian B, Fonarow GC. Epidemiology and aetiology of heart failure. Nat Rev Cardiol, 2016, 13: 368-378.
5. Yeung DKY, Leung SWS, Xu YC, Vanhoutte PM, Man RYK. Puerarin, an isoflavonoid derived from Radix puerariae, potentiates endothelium-independent relaxation via the cyclic AMP pathway in porcine coronary artery. Eur J Pharmacol, 2006, 552: 105-111.
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