MiR-411-5p Promotes Vascular Smooth Muscle Cell Phenotype Switch by Inhibiting Expression of Calmodulin Regulated Spectrin-Associated Protein-1
Author:
Affiliation:
1. Department of Cardiology, Zhongda Hospital of Southeast University Medical School
2. Medical School of Southeast University
Publisher
International Heart Journal (Japanese Heart Journal)
Link
https://www.jstage.jst.go.jp/article/ihj/65/3/65_23-590/_pdf
Reference31 articles.
1. 1. Shishido K, Antoniadis AP, Takahashi S, Tsuda M, Mizuno S, Andreou I, Papafaklis MI, Coskun AU, O'Brien C, Feldman CL, Saito S, Edelman ER, Stone PH. Effects of Low Endothelial Shear Stress After Stent Implantation on Subsequent Neointimal Hyperplasia and Clinical Outcomes in Humans. J Am Heart Assoc 2016; 5: e002949.
2. 2. Kapustin AN, Shanahan CM. Emerging roles for vascular smooth muscle cell exosomes in calcification and coagulation. J Physiol 2016; 594: 2905-14.
3. 3. Santulli G. microRNAs Distinctively Regulate Vascular Smooth Muscle and Endothelial Cells: Functional Implications in Angiogenesis, Atherosclerosis, and In-Stent Restenosis. Adv Exp Med Biol 2015; 887: 53-77.
4. 4. Zhang J, Gao F, Ni T, Lu W, Lin N, Zhang C, Sun Z, Guo H, Chi J. Linc-POU3F3 is overexpressed in in-stent restenosis patients and induces VSMC phenotypic transformation via POU3F3/miR-449a/KLF4 signaling pathway. Am J Transl Res 2019; 11: 4481-90.
5. 5. Yu X, Odenthal M, Fries JW. Exosomes as miRNA Carriers: Formation-Function-Future. Int J Mol Sci 2016; 17: 2028.
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