Tor Pathway Regulates Rrn3p-dependent Recruitment of Yeast RNA Polymerase I to the Promoter but Does Not Participate in Alteration of the Number of Active Genes
Author:
Affiliation:
1. Department of Biological Chemistry, University of California, Irvine, Irvine, California 92697-1700
2. Department of Microbiology, University of Virginia Health System, Charlottesville, Virginia 22908-0734
Abstract
Publisher
American Society for Cell Biology (ASCB)
Subject
Cell Biology,Molecular Biology
Reference54 articles.
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2. TOR controls translation initiation and early G1 progression in yeast.
3. Bodem, J., Dobreva, G., Hoffmann-Rohrer, U., Iben, S., Zentgraf, H., Delius, H., Vingron, M., and Grummt, I. (2000). TIF-IA, the factor mediating growth-dependent control of ribosomal RNA synthesis, is the mammalian homolog of yeast Rrn3p.EMBO Rep.1, 171-175.
4. Broach, J.R. (1991). RAS genes inSaccharomyces cerevisiae: signal transduction in search of a pathway.Trends Genet.7, 28-33.
5. Brun, R.P., Ryan, K., and Sollner-Webb, B. (1994). Factor C*, the specific initiation component of the mouse RNA polymerase I holoenzyme, is inactivated early in the transcription process.Mol. Cell. Biol.14, 5010-5021.
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