Contributions of Ccr4 and Gcn2 to the Translational Response of <i>C. neoformans</i> to Host-Relevant Stressors and Integrated Stress Response Induction-Reference-Cited by-同舟云学术

Contributions of Ccr4 and Gcn2 to the Translational Response of C. neoformans to Host-Relevant Stressors and Integrated Stress Response Induction

Published:2023-04-25 Issue:2 Volume:14 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Knowles Corey M.¹,Goich David¹,Bloom Amanda L. M.¹,Kalem Murat C.¹^ORCID,Panepinto John C.¹^ORCID

Affiliation:

1. Department of Microbiology and Immunology, Witebsky Center for Microbial Pathogenesis and Immunology, Jacobs School of Medicine and Biomedical Sciences, University at Buffalo, Buffalo, New York, USA

Abstract

Cryptococcus neoformans is a human pathogen capable of causing devastating infections. It must rapidly adapt to changing environments as it leaves its niche in the soil and enters the human lung.

Funder

Division of Extramural Research, National Institute of Allergy and Infectious Diseases

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mbio.00196-23

Reference63 articles.

1. Global burden of disease of HIV-associated cryptococcal meningitis: an updated analysis

2. Glucan Unmasking Identifies Regulators of Temperature-Induced Translatome Reprogramming in C. neoformans

3. Thermotolerance in the pathogen Cryptococcus neoformans is linked to antigen masking via mRNA decay-dependent reprogramming

4. Uncoupling of mRNA synthesis and degradation impairs adaptation to host temperature inCryptococcus neoformans

5. Translational Regulation Promotes Oxidative Stress Resistance in the Human Fungal Pathogen Cryptococcus neoformans

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1. MARCH2, a T cell specific factor that restricts HIV-1 infection;PLOS Pathogens;2024-07-29

2. Gcn2 rescues reprogramming in the absence of Hog1/p38 signaling inC. neoformansduring thermal stress;2024-06-11