Prevalence and Characterization of the Mechanisms of Macrolide, Lincosamide, and Streptogramin Resistance in Isolates of Streptococcus pneumoniae-Reference-Cited by-同舟云学术

Prevalence and Characterization of the Mechanisms of Macrolide, Lincosamide, and Streptogramin Resistance in Isolates of Streptococcus pneumoniae

Published:1998-09 Issue:9 Volume:42 Page:2425-2426
ISSN:0066-4804
Container-title:Antimicrobial Agents and Chemotherapy
language:en
Short-container-title:Antimicrob Agents Chemother

Author:

Johnston Nicole J.¹,de Azavedo Joyce C.¹,Kellner James D.²,Low Donald E.¹

Affiliation:

1. Department of Microbiology, Mount Sinai and Princess Margaret Hospitals, and the University of Toronto, Toronto, Ontario,1 and

2. Alberta Children’s Hospital and the University of Calgary, Calgary, Alberta,2 Canada

Abstract

ABSTRACT Of a total of 147 erythromycin-resistant Streptococcus pneumoniae isolates, 64 (43.5%) were resistant to erythromycin, clindamycin, and streptogramin B (MLS B phenotype), 57 of which possessed the ermB gene. Eighty-two (55.8%) were resistant to erythromycin alone (M phenotype), 81 of which possessed the mefE gene. One was erythromycin and streptogramin B resistant but susceptible to clindamycin (MS phenotype) and possessed neither the erm gene nor the mefE gene.

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Pharmacology (medical),Pharmacology

Link

https://journals.asm.org/doi/pdf/10.1128/AAC.42.9.2425

Reference10 articles.

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