Association of TRAF1/C5 Locus Polymorphisms with Epilepsy and Clinical Traits in Mexican Patients with Neurocysticercosis

Author:

Villegas Marcela1,Sciutto Edda2,Rosetti Marcos23,Fleury Agnes24,Fragoso Gladis2

Affiliation:

1. Facultad de Medicina Veterinaria y Zootecnia, Universidad Nacional Autónoma de México, Mexico City, Mexico

2. Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México, Mexico City, Mexico

3. Unidad Psicopatología y Desarrollo, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz, Mexico City, Mexico

4. Unidad Periférica del Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México en el Instituto Nacional de Neurología y Neurocirugía Dr. Manuel Velasco Suárez, Mexico City, Mexico

Abstract

Neurocysticercosis is caused by the establishment of Taenia solium cysts in the central nervous system. Murine cysticercosis by Taenia crassiceps is a useful model of cysticercosis in which the complement component 5 (C5) has been linked to infection resistance/permissiveness.

Funder

Consejo Nacional de Ciencia y Tecnología

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Immunology,Microbiology,Parasitology

Cited by 8 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. The role of Toll‐like receptors in neuropsychiatric disorders: Immunopathology, treatment, and management;Medicinal Research Reviews;2024-01-16

2. Host immune responses to Taenia infection;Molecular Medical Microbiology;2024

3. Epileptogenesis in Common Parasitic Infections;Current Neurology and Neuroscience Reports;2022-03-25

4. Taeniasis and Cysticercosis;Helminth Infections and their Impact on Global Public Health;2022

5. Interaction Between the Complement System and Infectious Agents – A Potential Mechanistic Link to Neurodegeneration and Dementia;Frontiers in Cellular Neuroscience;2021-08-02

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