Comparative Study of Virulence Traits of Escherichia coli Clinical Isolates Causing Early and Late Neonatal Sepsis
Author:
Affiliation:
1. Microbiology Department, Hospital Clinic, IDIBAPS, School of Medicine, University of Barcelona, Barcelona, Spain
Abstract
Publisher
American Society for Microbiology
Subject
Microbiology (medical)
Link
https://journals.asm.org/doi/pdf/10.1128/JCM.01682-07
Reference11 articles.
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2. Bingen, E., B. Picard, N. Brahimi, S. Mathy, P. Desjardin, J. Elion, and E. Denamur. 1998. Phylogenetic analysis of Escherichia coli strains causing neonatal meningitis suggests horizontal gene transfer from a predominant pool of highly virulent B2 group strains. J. Infect. Dis.177:642-650.
3. Rapid and Simple Determination of the Escherichia coli Phylogenetic Group
4. Huang, S., Y. Chen, G. Kong, S. H. M. Chen, J. Besemer, M. Borodovsky, and A. Jong. 2001. A novel genetic island of meningitic Escherichia coli K1 containing the ibeA invasion gene (GimA): functional annotation and carbon-source-regulated invasion of human brain microvascular endothelial cells. Funct. Integr. Genomics1:312-322.
5. Johnson, J. R., and A. Stell. 2000. Extended virulence genotypes of Escherichia coli strains from patients with urosepsis in relation to phylogeny and host compromise. J. Infect. Dis.181:261-272.
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