A Nine-Codon Deletion Mutation in the Cytomegalovirus UL97 Phosphotransferase Gene Confers Resistance to Ganciclovir
Author:
Affiliation:
1. Medical and Research Services, Veterans Affairs Medical Center, and Division of Infectious Diseases, Oregon Health Sciences University, Portland, Oregon
Abstract
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology
Link
https://journals.asm.org/doi/pdf/10.1128/AAC.44.1.183-185.2000
Reference15 articles.
1. A three-nucleotide deletion in the UL97 open reading frame is responsible for the ganciclovir resistance of a human cytomegalovirus clinical isolate
2. The Cys607→Tyr Change in the UL97 Phosphotransferase Confers Ganciclovir Resistance to Two Human Cytomegalovirus Strains Recovered from Two Immunocompromised Patients
3. Antiviral drug resistance in human cytomegalovirus.;Chou S.;Transplant. Infect. Dis.,1999
4. Analysis of the UL97 phosphotransferase coding sequence in clinical cytomegalovirus isolates and identification of mutations conferring ganciclovir resistance.;Chou S.;J. Infect. Dis.,1995
5. Frequency of UL97 phosphotransferase mutations related to ganciclovir resistance in clinical cytomegalovirus isolates.;Chou S.;J. Infect. Dis.,1995
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