Affiliation:
1. Institute of Medical Microbiology, University of Göttingen, Göttingen, Germany
Abstract
ABSTRACT
Microsporidia of the genus
Encephalitozoon
develop inside a parasitophorous vacuole (PV) of unknown origin. Using colocalization studies, the PV was found to be absent from the endocytic pathway markers early endosomal autoantigen 1, transferrin receptor, and lysosome-associated membrane protein 1 and for the endoplasmic reticulum marker calnexin. The nonfusiogenic characteristic of the PV appears to be acquired as early as 1 min postinfection and is not reversed by drug treatment with albendazole or fumagillin.
Publisher
American Society for Microbiology
Subject
Molecular Biology,General Medicine,Microbiology
Reference18 articles.
1. Bigliardi, E., and L. Sacchi. 2001. Cell biology and invasion of the microsporidia. Microbes Infect.3:373-379.
2. Developmental Expression of a Tandemly Repeated, Glycine- and Serine-Rich Spore Wall Protein in the Microsporidian Pathogen
Encephalitozoon cuniculi
3. Cali, A., and P. M. Takvorian. 1999. Developmental morphology and life cycles of the microsporidia, p. 85-128. In M. Wittner and L. M. Weiss (ed.), The microsporidia and microsporidiosis. American Society for Microbiology, Washington, D.C.
4. Phagocytic Uptake of
Encephalitozoon cuniculi
by Nonprofessional Phagocytes
5. Foucault, C., and M. Drancourt. 2000. Actin mediates Encephalitozoon intestinalis entry into the human enterocyte-like cell line, Caco-2. Microb. Pathog.28:51-58.
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