Sp1 Phosphorylation and Its Regulation of Gene Transcription
Author:
Affiliation:
1. Centre for Vascular Research, University of New South Wales, Sydney, Australia
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.01828-08
Reference74 articles.
1. Armstrong, S. A., D. A. Barry, R. W. Leggett, and C. R. Mueller. 1997. Casein kinase II-mediated phosphorylation of the C terminus of Sp1 decreases its DNA binding activity. J. Biol. Chem.272:13489-13495.
2. Bonello, M. R., Y. V. Bobryshev, and L. M. Khachigian. 2005. Peroxide-inducible Ets-1 mediates platelet-derived growth factor receptor-α gene transcription in vascular smooth muscle cells. Am. J. Pathol.167:1149-1159.
3. Bonello, M. R., and L. M. Khachigian. 2004. Fibroblast growth factor-2 represses platelet-derived growth factor receptor-alpha (PDGFR-alpha) transcription via ERK1/2-dependent Sp1 phosphorylation and an atypical cis-acting element in the proximal PDGFR-alpha promoter. J. Biol. Chem.279:2377-2382.
4. Briggs, M. R., J. T. Kadonaga, S. P. Bell, and R. Tjian. 1986. Purification and biochemical characterization of the promoter-specific transcription factor, Sp1. Science234:47-52.
5. Camitta, M. G., S. A. Gabel, P. Chulada, J. A. Bradbury, R. Langenbach, D. C. Zeldin, and E. Murphy. 2001. Cyclooxygenase-1 and -2 knockout mice demonstrate increased cardiac ischemia/reperfusion injury but are protected by acute preconditioning. Circulation104:2453-2458.
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