Long-Range Communication between the Silencers of HMR
Author:
Affiliation:
1. Department of MCD Biology, Sinsheimer Labs, 1156 High Street, University of California, Santa Cruz, California 95064
2. Department of Pharmacology, 683 Hoes Lane, UMDNJ-Robert Wood Johnson Medical School, Piscataway, New Jersey 08854
Abstract
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.01647-07
Reference63 articles.
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2. Ansari, A., and M. R. Gartenberg. 1999. Persistence of an alternate chromatin structure at silenced loci in vitro. Proc. Natl. Acad. Sci. USA96:343-348.
3. Ansari, A., and M. Hampsey. 2005. A role for the CPF 3′-end processing machinery in RNAP II-dependent gene looping. Genes Dev.19:2969-2978.
4. Aparicio, O. M., and D. E. Gottschling. 1994. Overcoming telomeric silencing: a trans-activator competes to establish gene expression in a cell cycle-dependent way. Genes Dev.8:1133-1146.
5. Blanton, J., M. Gaszner, and P. Schedl. 2003. Protein:protein interactions and the pairing of boundary elements in vivo. Genes Dev.17:664-675.
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