SLF2 Interacts with the SMC5/6 Complex to Direct Hepatitis B Virus Episomal DNA to Promyelocytic Leukemia Bodies for Transcriptional Repression-Reference-Cited by-同舟云学术

SLF2 Interacts with the SMC5/6 Complex to Direct Hepatitis B Virus Episomal DNA to Promyelocytic Leukemia Bodies for Transcriptional Repression

Published:2023-07-27 Issue:7 Volume:97 Page:
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:J Virol

Author:

Yao Qiyan¹²,Peng Bo²,Li Cong²,Li Xuelei²³,Chen Mingyi²,Zhou Zhongmin²,Tang Dingbin²,He Jiabei²,Wu Yumeng²,Sun Yinyan²,Li Wenhui²⁴^ORCID

Affiliation:

1. Graduate School of Peking Union Medical College, Chinese Academy of Medical Sciences, Beijing, China

2. National Institute of Biological Sciences, Beijing, China

3. School of Life Sciences, Tsinghua University, Beijing, China

4. Tsinghua Institute of Multidisciplinary Biomedical Research, Tsinghua University, Beijing, China

Abstract

Chronic HBV infection remains a major public health problem worldwide. Current antiviral treatments rarely cure the infection, as they cannot clear the viral reservoir, cccDNA, in the nucleus.

Funder

MOST | National Natural Science Foundation of China

Ministry of Science and Technology of the People's Republic of China

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/jvi.00328-23

Reference49 articles.

1. Molecular biology of hepatitis B virus infection

2. Global prevalence, treatment, and prevention of hepatitis B virus infection in 2016: a modelling study

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5. HBV cccDNA: viral persistence reservoir and key obstacle for a cure of chronic hepatitis B

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