Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics-Reference-Cited by-同舟云学术

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Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics

Published:2015-10 Issue: Volume:122 Page:91-100
ISSN:0166-3542
Container-title:Antiviral Research
language:en
Short-container-title:Antiviral Research

Author:

Guo Ju-Tao,Guo Haitao

Funder

NIH

Indiana University

Publisher

Elsevier BV

Subject

Virology,Pharmacology

Reference139 articles.

1. Half-life of the duck hepatitis B virus covalently closed circular DNA pool in vivo following inhibition of viral replication;Addison;J. Virol.,2002

2. Proteolytic disassembly is a critical determinant for reovirus oncolysis;Alain;Mol. Ther.,2007

3. Efficient persistence of extrachromosomal KSHV DNA mediated by latency-associated nuclear antigen;Ballestas;Science,1999

4. The amino-terminal domain of the hepadnaviral P-gene encodes the terminal protein (genome-linked protein) believed to prime reverse transcription;Bartenschlager;EMBO J.,1988

5. Nuclear HBx binds the HBV minichromosome and modifies the epigenetic regulation of cccDNA function;Belloni;Proc. Natl. Acad. Sci. U.S.A.,2009

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2. Tripartite Motif-Containing Protein 65 (TRIM65) Inhibits Hepatitis B Virus Transcription;Viruses;2024-05-31

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