Contribution of Candida albicans ALS1 to the Pathogenesis of Experimental Oropharyngeal Candidiasis
Author:
Affiliation:
1. Biological Research Laboratories
2. Laboratory Animal Science and Toxicology Research Laboratories, Sankyo Co., Ltd., Shinagawa-ku, Tokyo 140-8710, Japan
3. Harbor-UCLA Research and Education Institute, Torrance, California 90502
Abstract
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.70.9.5256-5258.2002
Reference12 articles.
1. Braun, B. R., and A. D. Johnson. 2000. TUP1, CPH1 and EFG1 make independent contributions to filamentation in Candida albicans. Genetics155:57-67.
2. Brown, A. J., and N. A. Gow. 1999. Regulatory networks controlling Candida albicans morphogenesis. Trends Microbiol.7:333-338.
3. Isogenic strain construction and gene mapping in Candida albicans.
4. Expression of the Candida albicans Gene ALS1 in Saccharomyces cerevisiae Induces Adherence to Endothelial and Epithelial Cells
5. Fu Y. A. S. Ibrahim D. C. Sheppard Y.-C. Chen S. W. French J. Cutler S. G. Filler and J. E. Edwards Jr. 2002. Candida albicans Als1p: an adhesin that is a downstream effector of the EFG1 filamentation pathway. Mol. Microbiol. 44 : 61-72.
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