PA Residues in the 2009 H1N1 Pandemic Influenza Virus Enhance Avian Influenza Virus Polymerase Activity in Mammalian Cells-Reference-Cited by-同舟云学术

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PA Residues in the 2009 H1N1 Pandemic Influenza Virus Enhance Avian Influenza Virus Polymerase Activity in Mammalian Cells

Published:2011-05-11 Issue:14 Volume:85 Page:7020-7028
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:Journal of Virology

Author:

Bussey K. A.,Desmet E. A.,Mattiacio J. L.,Hamilton A.,Bradel-Tretheway B.,Bussey H. E.,Kim B.,Dewhurst S.,Takimoto T.

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Reference36 articles.

1. The human H5N1 influenza A virus polymerase complex is active in vitro over a broad range of temperatures, in contrast to the WSN complex, and this property can be attributed to the PB2 subunit;Bradel-Tretheway;J. Gen. Virol.,2008

2. PB2 residue 271 plays a key role in enhanced polymerase activity of influenza A viruses in mammalian host cells;Bussey;J. Virol.,2010

3. Emergence of a novel swine-origin influenza A (H1N1) virus in humans;Dawood;N. Engl. J. Med.,2009

4. The cap-snatching endonuclease of influenza virus polymerase resides in the PA subunit;Dias;Nature,2009

5. Differential polymerase activity in avian and mammalian cells determines host range of influenza virus;Gabriel;J. Virol.,2007

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