Cell Death in Escherichia coli: Incomplete Base Excision Repair under Depletion of DapB and Dxr Proteins-Reference-Cited by-同舟云学术

Cell Death in Escherichia coli: Incomplete Base Excision Repair under Depletion of DapB and Dxr Proteins

Published:2022-08-30 Issue:4 Volume:13 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Azizoglu Reha Onur¹^ORCID

Affiliation:

1. Department of Gene and Cell Therapy and Food Engineering, Mediterranean (Akdeniz) University, Antalya, Turkey

Abstract

The generation of reactive oxygen species (ROS) within the cell is a significantly shared aspect of bacterial cell death against different stress conditions. The main cell death mechanism due to the generation of reactive oxygen species is then the incomplete base excision repair (BER) in response to oxidized nucleotides.

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mbio.01611-22

Reference8 articles.

1. A Common Mechanism of Cellular Death Induced by Bactericidal Antibiotics

2. Lethality of MalE-LacZ hybrid protein shares mechanistic attributes with oxidative component of antibiotic lethality

3. Degradation of the Escherichia coli Essential Proteins DapB and Dxr Results in Oxidative Stress, which Contributes to Lethality through Incomplete Base Excision Repair

4. Incomplete base excision repair contributes to cell death from antibiotics and other stresses

5. NMR Studies Uncover Alternate Substrates for Dihydrodipicolinate Synthase and Suggest That Dihydrodipicolinate Reductase Is Also a Dehydratase

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