Activation of the glnA , glnK , and nac Promoters as Escherichia coli Undergoes the Transition from Nitrogen Excess Growth to Nitrogen Starvation
Author:
Affiliation:
1. Department of Biological Chemistry, University of Michigan Medical School, Ann Arbor, Michigan 48109
2. Department of Biochemistry and Molecular Biology, Wayne State University School of Medicine, Detroit, Michigan 48201
Abstract
Publisher
American Society for Microbiology
Subject
Molecular Biology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JB.184.19.5358-5363.2002
Reference27 articles.
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2. Atkinson, M. R., and A. J. Ninfa. 1998. Role of the GlnK signal transduction protein in the regulation of nitrogen assimilation in Escherichia coli. Mol. Microbiol.29:431-447.
3. Backman, K., Y.-M. Chen, and B. Magasanik. 1981. Physical and genetic characterization of the glnA-glnG region of the Escherichia coli chromosome. Proc. Natl. Acad. Sci. USA78:3743-3747.
4. Carmona, M., F. Claverie-Martin, and B. Magasanik. 1997. DNA bending and the initiation of transcription at σ54-dependent bacterial promoters. Proc. Natl. Acad. Sci. USA94:9568-9572.
5. A method for constructing single-copy lac fusions in Salmonella typhimurium and its application to the hemA-prfA operon
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