Does TFAP2C govern conflicting cell fates in mouse preimplantation embryos?-Reference-Cited by-同舟云学术

Does TFAP2C govern conflicting cell fates in mouse preimplantation embryos?

Published:2024-01-02 Issue:4 Volume:167 Page:
ISSN:1470-1626
Container-title:Reproduction
language:
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Author:

Driscoll Chad S¹,Kim Jaehwan¹²,Ashry Mohamed¹,Knott Jason G¹^ORCID

Affiliation:

1. Department of Animal Science, Developmental Epigenetics Laboratory, Reproductive and Developmental Sciences Program, Michigan State University, East Lansing, Michigan, USA

2. Department of Animal Sciences, University of Missouri, Columbia, Missouri, USA

Abstract

Transcription factor AP2 gamma (TFAP2C) is a well-established regulator of the trophoblast lineage in mice and humans, but a handful of studies indicate that TFAP2C may play an important role in pluripotency. Here, we hypothesize and provide new evidence that TFAP2C functions as an activator of trophoblast and pluripotency genes during preimplantation embryo development.

Publisher

Bioscientifica

Link

https://rep.bioscientifica.com/downloadpdf/journals/rep/167/4/REP-23-0440.xml

Reference14 articles.

1. Transcription factor AP-2γ induces early Cdx2 expression and represses HIPPO signaling to specify the trophectoderm lineage;Cao,2015

2. The TFAP2C-regulated OCT4 naive enhancer is involved in human germline formation;Chen,2018

3. Glycolysis-independent glucose metabolism distinguishes TE from ICM fate during mammalian embryogenesis;Chi,2020

4. Transcription factor AP-2γ is a core regulator of tight junction biogenesis and cavity formation during mouse early embryogenesis;Choi,2012

5. Evidence that transcription factor AP-2γ is not required for Oct4 repression in mouse blastocysts;Choi,2013

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